Naturally I have an opinion about this, being that I am one of the great medical minds of the 17th century. I would say that you stand a good chance of catching "mad cow" disease, if in fact you don't already have it.....See your physician immediately, and give your possessions to the poor, or to me if you wish for distribution of course....
By far the largest ingestion of aluminum, for most people in the US, is due to baking powder. It's harmless. Although Alzheimer victims have been found to have higher than average Al content in there brains, current thinking is that the higher levels are only incidental to Alzheimers. Alzheimers victims have incorrect, beta amyloid folding of proteins in the brain and current research is centered on that. Small, low level, exposure to mercury metal or amalgams also seems to be harmless. Mercury is eliminated through the kidneys, unlike lead which tends to persist as insoluble phosphate. Many millions have amalgam fillings and mental acuity and health has improved dramatically since amalgam began to be used for fillings. Drawing the standard junk science conclusion from that fact, one concludes that mercury makes people brighter and healthier. Exposure to toxic levels of either mercury vapor, which will not occur at room temperature even if one works daily over a pool of mercury metal, or exposure to toxic levels of organic mercury compounds is quite a different matter. The most careful study of lead exposure, the results of which were published in the NIH Journal some years ago, showed that the safe levels were considerably above what is thought. The study has been ignored. Everyone who grew up in the age of leaded gasoline had, and has, what by today's standards are very high lead levels. There is no evidence that that generation, on average, was mentally deficient compared to prior or more recent generations. There is some evidence to suggest that Einstein, Feinman, and Fermi were not especially dull witted. Drawing the typical junk science conclusion from that fact, one concludes that exposure to lead causes mental brilliance. The hypothesis that lead exposure may result in delayed mental deficiency is interesting, and perhaps worth further study. Since in the popular press the levels of significance associated with results from controlled studies are seldom stated, one has to wonder what those levels were in the present study of delayed effects of exposure to heavy metals. One would certainly want to consider that before sinking a lot of money into further study.
I think you will find the expression "mad as a hatter" comes from the behaviour of people exposed to mercury salts over a lifetime in the hat making industry where it was used in the treatment of felt. Like lead it is a cumulative poison and is NOT readily excreted.
Talk of clinical trials of a preventative for alzheimers here (UK) in the press last week or so.An anti-oxidant made small enough to pass through the blood-brain barrier.Works in animals,please God it will work in humans.
It's copper you need to worry about not Al Clinical Correlation of Copper and Alzheimer's Disease Over the last several years, an increasing body of evidence points to dysfunctional copper homeostasis in the pathogenesis of AD. In 2006, a published epidemiological study conducted in 3718 patients over 65 years of age over a six year period, found that <B>subjects that consumed a typical amount of a copper supplement (1.6mg of copper a day) when taken together with a high saturated and trans fat diet had an equivalent 19 years of cognitive decline over such six year period as compared to those who did not consume such copper</B> (p < 0.001) (1). In a separate clinical study conducted by Prof. Rossini and Dr. Squitti in 53 patients, correlated the levels of the highly reactive 'free' copper (also known as copper not bound to ceruloplasmin) pool in serum to disease severity in AD patients versus aged-matched control patients. These results demonstrated that the 'free' copper serum pool (p < 0.0001) was highly increased in AD patients (2). http://www.reuters.com/article/pressRelease/idUS116736+07-Jan-2008+MW20080107 Jan 9th 2008 Buy Pipex stock if you fancy a trade on this, their Coprexa drug looks to help this
Alternatively, Turmeric (an Indian spice) Low incidences of Alzheimners in areas of India where it is a major art of the diet. Inhibitory action on Amyloid Plaque formation confirmed in lab studies. Sprinkle a teaspoon on your meusli in the morning.
Sorry, but you are only partially correct. Mercury poisoning is most definitely NOT cumulative. The hatters were exposed to chronically high levels becausethey used mercuric nitrate on their brushes and were in the habit of using saliva to create a fine point on the brush tip. Their exposure to soluble mercury II ion was high and daily. Mercury is Not a cumulative poison. Enough of Junk, popular press science! I can site two specific cases, although there is a plethora of lab work that shows mercury to be readily excreted. In one case, when the old electrochemistry laboratory at Cambridge was dismantled and the wooden floor taken up it was discovered that workers had been working for years directly over a pool of metallic mercury. Naturally this raised concerns regarding chronic exposure to mercury vapor. When these workers were tested they showed no higher levels then in the general populace. Though they were exposed to elevated vapor levels daily, their excretion rate was sufficient to bring them after a few weeks to normal levels. Had Mercury vapor been a cumulative poison they of course would have had permanently elevated levels. It is extremely easy to demonstrate that mercury is not a cumulative poison using laboratory animals. In Roswell New Mexico, a family was seriously poisoned after eating a pig that they had feed on grain treated with a mercurial fungacide. They were hospitalized and all nearly died and showed neurological effects. They all excreted excessive mercury. All but one, who died from acute poisoning, made a full recovery. When considering mercury exposure, what is important is the level of exposure and the rate of exposure.