the sooner we get away from BEEF

It was me who said that.

Try eating a meat only diet. You can't get fat that way.

So it must be the veges, fruits and grains!

 

Yes, most omnivores eat too many starchy vegetables, fruits and processed grain products which tends to make them overweight and raises their BP.

They don't get high BP from eating meat.

 

Too many calories make you fat. BEEF is high in fat and calories. Saturated fat raises LDL. High LDL hardens the arteries.

You do the math

 

i'm so confused by what to eat. I actually went vegetarian for 6 months, but in the end i decided i feel better when i eat some meat. currently i eat about 1 lb. of meat each day, tons of fresh vegetables and fresh fruits and some whole grains.

I only weigh about 160 lbs. and i feel really good except for when i go on a massive eating binge. that's when i feel like i damage myself. today i was stressed out after the trading day, and I couldn't stop eating. 3 large cheese quesadillas later i felt like i was going to pop.

i know cheese isn't very good for me. i'm not going to buy any more b/c when it is in the fridge i put it on everything. cheese is my favorite food probably.

i'm a firm believer that good health is vital to good trading.

 

I would think most people who are fat eat a lot of junk like cookies, cake, chips, ice cream, pizza and drink soft drinks or beer, ect. I doubt if they got fat from eating beef.

 

It really doesnt matter what you eat, as long as you drink a glass or 2 of red wine with your meal.

 

http://bacteriality.com/2008/01/26/cad/

There’s also no denying that cholesterol levels tend to rise in people with CAD. The question is why. Do cholesterol levels rise because of the fact that people are eating more of the substance, thus clogging their arteries? Or does cholesterol rise simply as a result of the disease process? Could it be that high cholesterol is simply a sign that a person is accumulating the L-form bacteria responsible for causing heart disease, just as low levels of the vitamin D metabolite 25-D in patients with most chronic diseases is not a sign of deficiency but rather an indication of L-form bacterial infection and immune dysfunction?

“Whether they’re high or low, either our cholesterol levels directly increase mortality or they’re the symptom of an underlying disorder that itself increases our risk of disease,” states Taubes. Although not enough research has been done on the subject at the moment to arrive at a definite answer, the results of numerous studies show that the latter may very well be the case.

Over the years, multiple large studies have shown that reducing cholesterol levels in patients at risk for CAD has little, if any, effect on mortality. In 2001, the Cochrane Collaboration published a systematic review that analyzed 27 of the most rigorous studies to test the ability of reduced or modified diets to prevent cardiovascular disease. Together the trials accounted for around 10,000 subjects followed for an average of three years each.

“The review concluded that the diets, whether low fat or cholesterol lowering, had no effect on longevity and had no “significant effect on cardiovascular events.””The review concluded that the diets, whether low fat or cholesterol lowering, had no effect on longevity and had no “significant effect on cardiovascular events.” Five years later Cochrane published another review, which again examined multiple risk factors including lowering blood pressure and cholesterol in order to see if the substances affected heart disease and mortality. This time around they analyzed the results of 39 trials involving over 900,000 patients, and once again found that the “pooled effects suggest multiple risk factor intervention has no effect on mortality.”

Similarly, the results of the Framingham study, one of the largest studies on cardiovascular disease conducted to date, found that for both men and women over 50 years of age, life expectancy showed no association with cholesterol levels. The likelihood of suffering a fatal first heart attack was no less for those with a cholesterol level of 180 mg/dl than it was for those with 250. In fact, those Framingham residents whose cholesterol declined over the first 14 years of observation were more likely to die prematurely than those whose cholesterol remained the same or increased.

Then there’s the MONitoring heart disease study or MONICA conduced by the World Health Organization in the 1970s which has been described as “far and away the biggest international collaborative study of cardiovascular disease ever carried out.” By the late 1990s, MONICA had recorded 150,000 heart attacks and analyzed 180,000 risk factor records. The results? Heart disease mortality was declining worldwide but that decline was independent of cholesterol levels, blood pressure, or even smoking habits.

Many studies today continue to obtain the same results. Take. for example, a study conducted just a few months ago by researchers at the University of Oxford in Britain, whose study results indicated that lower cholesterol levels were not linked to reduced stroke deaths.[26] The team, which analyzed 61 previous studies involving almost 900,000 adults, conducted mostly in western Europe and North America, found no relationship between total cholesterol levels and risk of stroke death, especially at older ages and among people with higher blood pressures.

The article goes on the explain why statins remain effective against heart disease
(hint: it's not related to their ability to lower cholesterol)

 

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This is from your link Promagma. Inflammation have to be studied.
Inflammation is good to fight infection, but when it stay on all the time in low level, inflammation become bad. Why it does not turn off for some people?
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"Clearly in Eisenhower’s case, and in the case of many other people with coronary artery disease or CAD, something other than fat and cholesterol consumption drives progression of the disease. As Taubes describes it, the long held dogma about heart disease, in which cholesterol clogging the arteries and excess body fat are viewed as culprits, “as though the fat of a greasy hamburger were transported directly from the stomach to the artery lining,” needs major revision.

In a paper published in the New England Journal of Medicine, researcher Göran K. Hansson explains that while it was previously believed that heart disease was caused by progressive narrowing of the blood vessels as smooth-muscle cells gradually formed plaque, “Recent research has shown that inflammation plays a key role in coronary artery disease and other manifestations of atherosclerosis.”[1]

 

Camel burger newest "healthy" option on Dubai menu

http://news.yahoo.com/s/nm/20100204/od_nm/us_emirates_camel

sounds yummy

 

http://www.youtube.com/watch?v=9gTLpTq1nQk&feature=player_embedded

Calorie Density with Jeff Novick