I don't know that this is going to happen for sure... but I think you may be correct. However, so much of Northern CA and parts of San Diego are tech related... I think CA may do O.K. after a longer period of time to recovery. L.A. may be screwed for a while.
So far, Governor Gavin Newsom announced 4.3 million in actual job losses, a $54 billion deficit, refused to pay $2.4 billion in obligations to fund state workers pensions, 10% pay cut for all state workers. Also, he set aside $72 million just to give to illegals. We have not even gotten to the tax increases coming in vehicle registration fees, sales taxes, gasoline taxes, property taxes and whatever taxes he decides to impose. With the State Capitol in full control of the Democrats, they will just pass anything he wants. At the local level, Mayor Eric Garcetti furloughed city employees the equivalent of 1 whole month's salary for starters so that, he can fund the Angeleno Card targeted towards illegals, it will provide $1,500 to cardholders monthly. Hell, that is way more than the $1,200 stimulus we got from the Federal government. This Angeleno card is open ended and no indication of when it will end. In other news, Mayor Eric Garcetti announced he will strip $150 million from the Los Angeles Police Department's budget in addition to ordering the police not to crackdown on gang members. Good job these Democrat Mayors and Governors doing to destroy their cities and states.
You've spammed the board repetitively with your opinion on the virus for months. Verbose, rambling like a mad man, pushing a selfish attitude. So you can't deal with some backlash, too bad. Fuck off. Society may not be able to stop idiots like you from spreading the disease but we can damn well tell you you're a fool for doing it.
I responded your nasty post to me. I was not even conversing with you. Apparently you are the one who can't handle it. My attitude is not selfish at all. the problem is simply minded fools don't realize that there was no science behind the lock down of low risk groups past the first 2 to 3 weeks. And there are some models showing the lock down does more harm do good. So quite the contrary... imo its you scared SOBS who are being the selfish fools for the last 60 days. There is no science or data... showing the shutdown of low risk groups does any good after the hospital beds are empty. you have the selfish attitude. (By the way, I made seven figures during the last mortgage crisis. The longer they shutdown the economy... the more money I will likely make. )
Here are the states who according to the left were "selfish" and killing themselves to by reopening early. It was not selfish to understand that the models the experts and the left were touting were not based on science and data. They expected disaster. So are its not happening. My point on this thread and every other is that... the shutdown of low risk groups was not scientific after the first 3 weeks. its not selfish... to desire what is best for others.
Another "Surprise for the narrative. We may not all be equally equally at risk to the virus. - Some People May Have a Head Start Against Coronavirus, Surprising Evidence Shows AYLIN WOODWARD, BUSINESS INSIDER 4 JUNE 2020 Some people's immune systems may have a head start in fighting the coronavirus, recent research suggested. A study published last month in the journal Cell showed that some people who have never been exposed to the coronavirus have helper T cells that are capable of recognising and responding to it. The likeliest explanation for the surprising finding, according to the researchers, is a phenomenon called cross-reactivity: when helper T cells developed in response to another virus react to a similar but previously unknown pathogen. In this case, those T cells may be left over from people's previous exposure to a different coronavirus – likely one of the four that cause common colds. "You're starting with a little bit of an advantage – a head start in the arms race between the virus that wants to reproduce and the immune system wanting to eliminate it," Alessandro Sette, one of the study's coauthors, told Business Insider. He added that cross-reactive helper T cells could "help generate a faster, stronger immune response." An immunological 'head start' For its study, Sette's team examined the immune systems of 20 people who got the coronavirus and recovered, as well as blood samples from 20 people that had been collected between 2015 and 2018 (meaning there was no chance those people had been exposed to the new coronavirus). Among the 20 people whose blood samples were taken before the pandemic, 50 percent had a type of white blood cell called CD4+ – T cells that help the immune system create antibodies – that the researchers found to be capable of recognising the new coronavirus and prompting the immune system to fight back right away. More research is needed to know whether or to what degree this cross-reactivity influences the severity of a case. "It is too early to conclude that cross-reactivity with cold coronaviruses plays a role in the mild or severe clinical outcome of COVID-19 or the degree of infection in the populations," Maillère Bernard, a scientist at CEA/Université de Paris-Saclay in France who was not involved in the study, told Business Insider. Evidence for immunity Among the group of coronavirus patients studied in the new research, only two had severe cases; the other 90 percent had either mild or moderate infections. The group was selected that way so that the researchers could measure immune responses in average COVID-19 patients, not hospitalized people. (An estimated 20 percent of coronaviruses cases are severe.) "If you're looking at the exception rather than rule, it's hard to know what's going on," Crotty said. "If the average immune response looked terrible, it would be a big red flag." The researchers searched the patients' blood for two types of white blood cells: CD4+ cells and CD8+ cells, which are killer T cells that attack virus-infected cells. The results showed that during the course of their infections, all 20 patients made antibodies and helper T cells capable of recognising the coronavirus and responding accordingly, and 70 percent made killer T cells. This suggests the body will be able to identify and defend itself against the coronavirus in the future. "Obviously we cannot tell you with a straight face what will happen 15 years from now because the virus has only been around for a few months. So nobody knows whether this immune response is long-lived or not," Sette said. But he thinks there's reason for optimism, especially for patients who had severe cases. "The immune memory is related to the event. If it's a strong event, you'll have a strong memory," Sette added. "If you almost got run over by a truck, you'll remember it, but you may not remember the colour of the socks you wore yesterday because it's not a big deal." Yuan Tian, a scientist at the Fred Hutch Institute in Seattle who was not involved in the research, told Business Insider that to learn more about how T cells relate to immunity, "it'd be interesting to study people with severe disease and compare the T-cell response between them and those with mild disease." That's next on the docket, according to Crotty. "We're looking to identify T-cell response in the critically hospitalized," he said. "It's being done as we speak." https://www.sciencealert.com/surpri...e-are-already-primed-to-fight-the-coronavirus
the link to the study... below... plus a very interesting paragraph discussing whether its possible some of us may have pre existing immunity. https://www.cell.com/cell/fulltext/S0092-8674(20)30610-3 While it was important to identify antigen-specific T cell responses in COVID-19 cases, it is also of great interest to understand whether cross-reactive immunity exists between coronaviruses to any degree. A key step in developing that understanding is to examine antigen-specific CD4+ and CD8+ T cells in COVID-19 cases and in unexposed healthy controls, utilizing the exact same antigens and series of experimental techniques. CD4+ T cell responses were detected in 40%–60% of unexposed individuals. This may be reflective of some degree of cross-reactive, preexisting immunity to SARS-CoV-2 in some, but not all, individuals. Whether this immunity is relevant in influencing clinical outcomes is unknown—and cannot be known without T cell measurements before and after SARS-CoV-2 infection of individuals—but it is tempting to speculate that the cross-reactive CD4+ T cells may be of value in protective immunity, based on SARS mouse models ( Zhao et al., 2016 ). Clear identification of the cross-reactive peptides, and their sequence homology relation to other coronaviruses, requires deconvolution of the positive peptide pools, which is not feasible with the cell numbers presently available, and time frame of the present study.