As explained multiple times already -- and backed by medical information -- there is no such thing as T-Cell Immunity for COVID-19. T-Cell resistance may reduce the severity of COVID-19 in people who are infected -- it in no way makes a person immune to COVID-19. Anyone pushing T-Cell immunity for COVID-19 is pushing a fairy tale. There is no herd immunity at 20 to 30 percent of the population for a highly infectious disease. The minimum level is 60% to 70% to achieve "herd immunity" from antibodies (and likely higher). You are peddling nonsense again.
Let's provide some more information about the recent study with some important points bolded and a link to the study... Coronavirus antibodies decline after infection, study finds, raising questions about herd immunity https://www.cnbc.com/2020/10/27/her...tibodies-fall-after-infection-study-says.html Researchers from Imperial College London screened 365,000 people in England over three rounds of testing between June 20 and September 28. Analysis of finger-prick tests carried out at home found that, rather than people building immunity over time, the number of people with antibodies that can fight Covid-19 declined roughly 26% over the study period. The findings suggest that there may be a decline in the level of population immunity in the months following the first wave of the coronavirus epidemic, potentially dashing the hopes of those calling for a controversial herd immunity response strategy. Antibodies against the coronavirus fall as people recover from the disease, according to the findings of a major U.K. study, potentially dealing a blow to those pushing for so-called herd immunity. Researchers from Imperial College London screened 365,000 people in England over three rounds of testing between June 20 and September 28. Analysis of finger-prick tests carried out at home found that, rather than people building immunity over time, the number of people with antibodies that can fight Covid-19 declined roughly 26% over the study period. The REACT-2 study, which has not yet been peer reviewed, found that 6% of those tested had antibodies to the virus when the U.K.’s lockdown measures were relaxed over the summer. However, by the start of the second wave of cases last month, this figure had fallen to 4.4%. “This very large study has shown that the proportion of people with detectable antibodies is falling over time,” said Helen Ward, one of the authors of the study and professor at Imperial College London. “We don’t yet know whether this will leave these people at risk of reinfection with the virus that causes COVID-19, but it is essential that everyone continues to follow guidance to reduce the risk to themselves and others.” What does it mean for herd immunity? The findings suggest that there may be a decline in the level of population immunity in the months following the first wave of the coronavirus epidemic, potentially dashing the hopes of those calling for a controversial herd immunity response strategy. Herd immunity occurs when enough of a population is immune to a disease, making it unlikely to spread and protecting the rest of the community, according to the Mayo Clinic. It can be achieved through natural infection — when enough people are exposed to the disease and develop antibodies against it — and through vaccinations. Health experts estimate that around 70% of the population would need to be vaccinated or have natural antibodies to achieve herd immunity. Some epidemiologists have suggested that aiming for herd immunity would be a better response to the pandemic than lockdown measures. Many others, however, have sharply criticized a strategy that could require vulnerable people to shield at home while the virus spreads through the young and healthy. Earlier this month, Dr. Anthony Fauci, the U.S.’s top infectious disease expert, described calls to let the virus rip through the U.S. population unchecked as “nonsense” and “dangerous.” To date, more than 43.5 million people around the world have contracted the coronavirus, with 1.16 million related deaths, according to data compiled by Johns Hopkins University. Implications for reinfection The results of the REACT-2 study showed a downward trend of antibodies in people of all age groups and in all areas of the U.K., but not in health workers. The decline was largest for people aged 75 and above, the study said, while the smallest fall was among those aged between 18 to 24 years old. Researchers found that the decline in prevalent antibodies may initially be rapid, before plateauing. They cautioned that data on this was only now beginning to emerge. (More at above url)
Where is your recent science you crack pot? I presented studies to you saying it is either an open question... or there seems to be a very good chance. like this one... why don't you try reading them... you covid fearful liar. https://www.contagionlive.com/view/emerging-data-support-lasting-covid19-immunity-t-cells These studies find t cell reactivity in the lab... and they say that based on the lack of evidence of people getting it a second time T cell immunity is very likely real. Then they quote scientists doing the research... “Memory T cells are known for their ability to affect the clinical severity and susceptibility to future infection,” authors write. “T cell studies documenting pre-existing reactivity to SARS-CoV-2 in 20-50% of people suggest that antibodies are not the full story.” So show us your science for the concept that there is no T cell immunity for Covid.
Go back and read the previous posts. At least you have become enough of a realist to at least shift your position to that T-Cell immunity for COVID is "an open question" from your previous claims that T-Cell immunity was absolutely going to provide herd immunity. I guess there is enough news out now that "T-Cell immunity" for COVID is a farce ---- that you are trying to re-position yourself.
I have never read a single article from you saying there is no such thing as T cell immunity. I just showed you that in the lab T cells are reacting with Covid and that the data in the population of small studies is suggesting T Cell immunity. You are defeated... What are you doing taking a victory lap you fraud. Post your data and science or shut up.
Immunity to the Coronavirus May Last Years, New Data Hint Apoorva Mandavilli 4 hrs ago Eight months after infection, most people who have recovered still have enough immune cells to fend off the virus and prevent illness, the new data show. A slow rate of decline in the short term suggests, happily, that these cells may persist in the body for a very, very long time to come. The research, published online, has not been peer-reviewed nor published in a scientific journal. But it is the most comprehensive and long-ranging study of immune memory to the coronavirus to date. That amount of memory would likely prevent the vast majority of people from getting hospitalized disease, severe disease, for many years,” said Shane Crotty, a virologist at the La Jolla Institute of Immunology who co-led the new study. The findings are likely to come as a relief to experts worried that immunity to the virus might be short-lived, and that vaccines might have to be administered repeatedly to keep the pandemic under control. And the research squares with another recent finding: that survivors of SARS, caused by another coronavirus, still carry certain important immune cells 17 years after recovering. The findings are consistent with encouraging evidence emerging from other labs. Researchers at the University of Washington, led by the immunologist Marion Pepper, had earlier shown that certain “memory” cells that were produced following infection with the coronavirus persist for at least three months in the body. A study published last week also found that people who have recovered from Covid-19 have powerful and protective killer immune cells even when antibodies are not detectable. These studies “are all by and large painting the same picture, which is that once you get past those first few critical weeks, the rest of the response looks pretty conventional,” said Deepta Bhattacharya, an immunologist at the University of Arizona. Akiko Iwasaki, an immunologist at Yale University, said she was not surprised that the body mounts a long-lasting response because “that’s what is supposed to happen.” Still, she was heartened by the research: “This is exciting news.” A small number of infected people in the new study did not have long-lasting immunity after recovery, perhaps because of differences in the amounts of coronavirus they were exposed to. But vaccines can overcome that individual variability, said Jennifer Gommerman, an immunologist at the University of Toronto. “That will help in focusing the response, so you don’t get the same kind of heterogeneity that you would see in an infected population,” she said. In recent months, reports of waning antibody levels have created worry that immunity to the coronavirus may disappear in a few months, leaving people vulnerable to the virus again. But many immunologists have noted that it is natural for antibody levels to drop. Besides, antibodies are just one arm of the immune system. Although antibodies in the blood are needed to block the virus and forestall a second infection — a condition known as sterilizing immunity — immune cells that “remember” the virus more often are responsible for preventing serious illness. “Sterilizing immunity doesn’t happen very often — that is not the norm,” said Alessandro Sette, an immunologist at the La Jolla Institute of Immunology and co-leader of the study. More often, people become infected a second time with a particular pathogen, and the immune system recognizes the invader and quickly extinguishes the infection. The coronavirus in particular is slow to do harm, giving the immune system plenty of time to kick into gear. “It may be terminated fast enough that not only are you not experiencing any symptoms but you are not infectious,” Dr. Sette said. Dr. Sette and his colleagues recruited 185 men and women, aged 19 to 81, who had recovered from Covid-19. The majority had mild symptoms not requiring hospitalization; most provided just one blood sample, but 38 provided multiple samples over many months. The team tracked four components of the immune system: antibodies, B cells that make more antibodies as needed; and two types of T cells that kill other infected cells. The idea was to build a picture of the immune response over time by looking at its constituents. “If you just look at only one, you can really be missing the full picture,” Dr. Crotty said. He and his colleagues found that antibodies were durable, with modest declines at six to eight months after infection, although there was a 200-fold difference in the levels among the participants. T cells showed only a slight, slow decay in the body, while B cells grew in number — an unexpected finding the researchers can’t quite explain. The study is the first to chart the immune response to a virus in such granular detail, experts said. “For sure, we have no priors here,” Dr. Gommerman said. “We’re learning, I think for the first time, about some of the dynamics of these populations through time.” Worries over how long immunity to the coronavirus persists were sparked mainly by research into those viruses causing common colds. One frequently cited study, led by Jeffrey Shaman of Columbia University, suggested that immunity might fade quickly and that reinfections could occur within a year. “What we need to be very mindful of is whether or not reinfection is going to be a concern,” Dr. Shaman said. “And so seeing evidence that we have this kind of persistent, robust response, at least to these time scales, is very encouraging.” So far, at least, he noted, reinfections with the coronavirus seem to be rare. Exactly how long immunity lasts is hard to predict, because scientists don’t yet know what levels of various immune cells are needed to protect from the virus. But studies so far have suggested that even small numbers of antibodies or T and B cells may be enough to shield those who have recovered. The participants in the study have been making those cells in robust amounts — so far. “There’s no sign that memory cells are suddenly going to plummet, which would be kind of unusual,” Dr. Iwasaki said. “Usually, there’s a slow decay over years.” There is some emerging evidence that reinfections with common cold coronaviruses are a result of viral genetic variations, Dr. Bhattacharya noted, and so those concerns may not be relevant to the new coronavirus. “I don’t think it’s an unreasonable prediction to think that these immune memory components would last for years,” he said. https://www.msn.com/en-us/health/me...irus-may-last-years-new-data-hint/ar-BB1b6dwt
Let's go to the underlying scientific paper. These people are merely talking their own book.. selling T-Cell remedies. https://www.biorxiv.org/content/10.1101/2020.11.15.383323v1 Competing Interest Statement A.S. is a consultant for Gritstone, Flow Pharma, Merck, Epitogenesis, Gilead and Avalia. S.C. is a consultant for Avalia. LJI has filed for patent protection for various aspects of T cell epitope and vaccine design work. Mount Sinai has licensed serological assays to commercial entities and has filed for patent protection for serological assays. D.S., F.A., V.S. and F.K. are listed as inventors on the pending patent application (F.K., V.S.), and Newcastle disease virus (NDV)-based SARS-CoV-2 vaccines that name F.K. as inventor. All other authors declare no conflict of interest. Of course.... Dr. Bhattacharya is a quack that pushes the Great Barrington Declaration and other nonsense -- is quoted in the article as well as his compatriots.