Certainly. I am actively trying to get it by being in large groups in order to see if airborne. Thus, I am performing an experiment as to whether or not it is airborne or by touching face. ---I already suspect it's not airborne so this is just a confirmation test.
HARD LUCK Covid patient suffers four-hour erection after coronavirus ‘triggered priapism and blood clots’ https://www.thesun.co.uk/news/12002...suffers-4-hour-erection-priapism-blood-clots/
Here is an interesting one www.bbc.com/news/health-53248660 Coronavirus: Immunity may be more widespread than tests suggest By Rachel SchraerHealth reporter 01 July 2020 Image copyrightSCIENCE PHOTO LIBRARY People testing negative for coronavirus antibodies may still have some immunity, a study has suggested. For every person testing positive for antibodies, two were found to have specific T-cells which identify and destroy infected cells. This was seen even in people who had mild or symptomless cases of Covid-19. But it's not yet clear whether this just protects that individual, or if it might also stop them from passing on the infection to others. Researchers at the Karolinksa Institute in Sweden tested 200 people for both antibodies and T-cells. Some were blood donors while others were tracked down from the group of people first infected in Sweden, mainly returning from earlier affected areas like northern Italy. This could mean a wider group have some level of immunity to Covid-19 than antibody testing figures, like those published as part of the UK Office for National Statistics Infection Survey, suggest. It's likely those people did mount an antibody response, but either it had faded or was not detectable by the current tests. And these people should be protected if they are exposed to the virus for a second time. Prof Danny Altmann at Imperial College London described the study as "robust, impressive and thorough" and said it added to a growing body of evidence that "antibody testing alone underestimates immunity" Herd immunity This doesn't necessarily get us any closer to herd immunity, though, according to assistant professor Marcus Buggert, one of the study's authors. More analysis needs to be done to understand whether these T-cells provide "sterilising immunity", meaning they completely block the virus, or whether they might protect an individual from getting sick but not stop them from carrying the virus and transmitting it. Much of the discussion around Covid-19 immunity has focused on antibodies - Y-shaped proteins which act like "missiles shooting down a target", assistant Prof Buggert explained. They bind to the virus before it can enter your cells, and neutralise it. If antibodies fail to neutralise the virus, it can enter your cells and turn them into virus-making factories. T-cells, on the other hand, target already-infected cells and completely destroy them, stopping them from spreading to other, healthy cells Like antibodies, T-cells are part of the bit of your immune system that has a memory. Once it recognises a particular virus, it can quickly target cells infected with it and kill them. T-cell crash Researchers from the Francis Crick Institute, King's College London and Guy's and St Thomas' Hospital noticed a group of 60 severely ill patients appeared to experience a crash in their numbers of T-cells. This was not observed in the Karolinska study, which found the sicker the patient, the higher the level of antibodies and T-cells they appeared to produce. The team said more research was needed. While theirs is the biggest T-cell study done so far, it still involved a relatively small group of patients. T-cells are very complex and much harder to identify than antibodies, requiring specialist labs and small batches of samples being tested by hand over the course of days. This means mass testing for T-cells is not a very likely prospect at the moment. Follow Rachel on Twitter Copyright © 2020 BBC. The BBC is not responsible for the content of external sites.
Boom... and just like that even my (and a few others) long shot theory is looking like it could turn out to be incredibly off the charts correct. Bugen even knows on other threads I said there may be other mechanisms in play besides antibodies. No worries lefties ... We don't expect any of you to apologize...
Yes. T cells and also"memory cells" are an underdiscussed part of the picture. The reporting on antibodies - ie. who has them and does not and therefore who is immune and who is not- is wayyyyyy too simplistic. As discussed, there is the issue of T-cells which may knock out the virus so fast that the body does not even gear up to produce a full blown colony of antibodies. But it would be a big mistake to think that that person is therefore highly vulnerable to covid. It's the opposite. In addition to antibodies, there are T-cells but there are also memory cells. These are the cells that become wired and coded to recognize a virus and remember it and are all titted up and ready to go when and if it reappears, so that the immune system does not fuck around for weeks trying to find the formula needed. They are the loaded revolver in the dresser draw who remembers you and will recognize you if your sorry arse comes back. So what happens is the patient recovers from a virus and eventually the antibodies peter out and a low antibody level shows in the blood and many conclude that the patient is no longer protected against reinfection. NOPE. This is another instance where that cannot necessarily be concluded because the patient is not back to square one. They have memory cells that are coded to produce antibodies much faster and more efficiently than the first time around. Further, and I discussed this wayy back along with the other factors, it appears (from studying the blood of fast responders) that many fast responders are able to knock out the covid virus with high levels of immunoglobulin G which is a common antibody which is also generated by other viruses such as influenza Type B. Bottom line is one cannot necessarily conclude that people without a high covid antibody level in their blood are highly vulnerable. Some are, but many are not because their immune system uses other pathways to make the kill and that can be a good thing. There is also an ongoing argument being made that if some people with covid become reinfected that we are all going to die because that proves that there is no immunity. NOPE. It just shows that some people may need another exposure to strengthen their immunity even more. We may get the flu a little bit each year but this just updates our immunity. Some people may say we are not protected if we still get the flu year after year. Really? Try infecting an aboriginal group that has been isolated and see what happens. The flu that may make you a little sick for a day or a few every year will wipe out their entire tribe. That's because you have basic immunity even though it needs to be updated. To my knowledge, almost all of the vaccine researchers are planning to introduce a vaccine that will require two shots spaced apart by a few weeks for all the reasons discussed above- programming the memory cells and giving some immunity so that you can give them an even large dose in the second shot. Out in nature, this is the equivalent of a person needing to be infected twice to get good immunity. It does not mean that they will not achieve immunity- although that may be true for a subset of people who are immunodeficient for the usual reasons- age, other illness etc. Alright nuff of that. but yes. there are t cells and memory cell programming and all sorts of other factors beyond just "have you got antibodies or not." although that is a good discussion too but kind of well worn compared to the other factors.
Your theory eh? yeah, boom. Well a lot of people (everyone pondering it?) have been wondering about what is driving the relatively mild response in many people. It is incredibly important for you to attach yourself to any credit for anything that might make you look better. You really should have become an economist.
Just shows to go how we all have the damned Influenza in us, all year long. It is just that after decades of vaccination campaigns, we have the "herd immunity". Very few of us fall prey to it in the summer because our immune-systems are strong. How about MMR vaccines? When was the last time one of you had the measles? We still give that vaccine to our kids, and should. That's why measles rarely breaks out these days. Folks, wear the mask, and get yer vaccines!
I thought flu was seasonal until I started living near the equator where there are no real seasons. You can get it any damn time of the year if you meet people who have been travelling just its more common around Christmas as people fly back from colder, usually more developed nations. Flu mutates a bit faster. I have to wonder with all these precautions will many variants for influenza A and B be extinct.